Rho-Kinase Mediates Angiotensin II-Induced Monocyte Chemoattractant Protein-1 Expression in Rat Vascular Smooth Muscle Cells
Por um escritor misterioso
Descrição
Agomelatine prevents angiotensin II-induced endothelial and mononuclear cell adhesion
Rho-kinase inhibition: a novel therapeutic target for the treatment of cardiovascular diseases. - Abstract - Europe PMC
Transmembrane signaling pathway mediates oxidized low-density lipoprotein-induced expression of plasminogen activator inhibitor-1 in vascular endothelial cells
Cardiac Hypertrophy: From Pathophysiological Mechanisms to Heart Failure Development
Angiotensin II Induces Monocyte Chemoattractant Protein-1 Gene Expression in Rat Vascular Smooth Muscle Cells
The Protective Effects of Tripeptides VPP and IPP against Small Extracellular Vesicles from Angiotensin II-Induced Vascular Smooth Muscle Cells Mediating Endothelial Dysfunction in Human Umbilical Vein Endothelial Cells
Angiotensin II cell signaling: physiological and pathological effects in the cardiovascular system
Frontiers Adiponectin Attenuates Angiotensin II-Induced Vascular Smooth Muscle Cell Remodeling through Nitric Oxide and the RhoA/ROCK Pathway
TRPM8 downregulation by angiotensin II in vascular smooth muscle cells is involved in hypertension
Frontiers An Insight on Multicentric Signaling of Angiotensin II in Cardiovascular system: A Recent Update
Frontiers Molecular Interactions Between Vascular Smooth Muscle Cells and Macrophages in Atherosclerosis
Rho kinase as a therapeutic target in cardiovascular disease
Rho-Kinase Mediates Angiotensin II-Induced Monocyte Chemoattractant Protein-1 Expression in Rat Vascular Smooth Muscle Cells
Angiotensin II-induced Hypertension is Reduced by Deficiency of P-selectin Glycoprotein Ligand-1
IJMS, Free Full-Text
de
por adulto (o preço varia de acordo com o tamanho do grupo)