Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN
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The Role of Mitochondria in the Immune Response in Critical Illness, Critical Care
Mitochondrial oxidative stress drives IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is controlled by Fas
Frontiers Mitochondria, Oxidative Stress and Innate Immunity
Progress in the Correlation Between Inflammasome NLRP3 and Liver Fibrosis
How cancer cells make and respond to interferon-I: Trends in Cancer
Reactive Oxygen Species: Drivers of Physiological and Pathological Pro
Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is regulated by Fas/FasL signaling
Journal of Cellular Physiology, Cell Biology Journal
Frontiers Reactive Oxygen Species in Macrophages: Sources and Targets
Mitochondrial dysfunction in fibrotic diseases
Increased mitochondrial fission induces NLRP3/cGAS-STING mediated pro-inflammatory pathways and apoptosis in UVB-irradiated immortalized human keratinocyte HaCaT cells - ScienceDirect
Mitochondrial Functions in Infection and Immunity: Trends in Cell Biology
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