CBP-HSF2 structural and functional interplay in Rubinstein-Taybi neurodevelopmental disorder
Por um escritor misterioso
Descrição
Stress pathways in neurodevelopmental disorders
PDF) Loss of CBP acetyltransferase activity by PHD finger mutations in Rubinstein-Taybi syndrome
Publications
Ubiquitination of HSF2 is enhanced by heat stress. (A) HEK293 cells
Gene expression changes in the transition from neural progenitors to
Proteomic analysis of 110A53T, A53T, and Hsp110 brains. (A) Synaptic
Deletion of Sse1 promotes Cin8-dependent spindle elongation in S phase.
CBP-HSF2 structural and functional interplay in Rubinstein-Taybi neurodevelopmental disorder
Immunofluorescence and morphological characterization of iPSC-derived
CBP-HSF2 structural and functional interplay in Rubinstein-Taybi neurodevelopmental disorder
The HDAC Inhibitor, TSA, Rescues the Decreased Cortical Precursor
CBP/EP300-dependent acetylation and stabilization of HSF2 are compromised in Rubinstein-Taybi syndrome
CBP/EP300-dependent acetylation and stabilization of HSF2 are compromised in Rubinstein-Taybi syndrome
LacZ expression as a reporter of the HSF2 expression pro®le. ( A and B
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