RVA-induced necrosome formation consisting of RIPK1, RIPK3, and MLKL
Por um escritor misterioso
Descrição
Post-translational control of RIPK3 and MLKL
The regulation of necroptosis by post-translational modifications
RIPK3-and MLKL-dependent necroptosis mediated by various stimuli.
Targeting Necroptosis as a Promising Therapy for Alzheimer's Disease
RIPK proteins in cell death and tissue homeostasis.
Necroptosis: a regulated inflammatory mode of cell death, Journal of Neuroinflammation
The Structural Basis of Necroptotic Cell Death Signaling: Trends in Biochemical Sciences
Bypassing drug resistance by triggering necroptosis: recent advances in mechanisms and its therapeutic exploitation in leukemia, Journal of Experimental & Clinical Cancer Research
RIP3 is an upregulator of aerobic metabolism and the enhanced respiration by necrosomal RIP3 feeds back on necrosome to promote necroptosis
Shikonin overcomes drug resistance and induces necroptosis by regulating the miR-92a-1-5p/MLKL axis in chronic myeloid leukemia
Frontiers Up-Regulation of RIP3 Alleviates Prostate Cancer Progression by Activation of RIP3/MLKL Signaling Pathway and Induction of Necroptosis
Apolipoprotein A1 Protects Against Necrotic Core Development in Atherosclerotic Plaques: PDZK1-Dependent High-Density Lipoprotein Suppression of Necroptosis in Macrophages
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